Multi-Hit Hypothesis for the Oncogenic Potential of mRNA COVID-19 Vaccines

Pathophysiologic Support for the Observation of "Turbo Cancer" after Vaccination

By PETER MCCULLOUGH, MD
SEP 29

By Peter A. McCullough, MD, MPH

As an internist and specialist, it is hard for me to believe a novel biologic product to cause heart, neurologic, thrombotic, and immunologic disease could also play a role in the initiation and acceleration of oncogenesis. In Western countries before the pandemic, the leading causes of death were heart disease 40%, cancer 40%, and death from other known causes (homicide, suicide, accidents, etc). The mRNA COVID-19 vaccines if proven to promote cancer, would then be implicated in rises in all-cause mortality being observed world wide.

In 1984, Sutherland and Bailer proposed the “Multi-Hit Hypothesis of Carcinogenesis:”

“A new multihit model of carcinogenesis is developed for use in evaluating age-specific cancer incidence rates in human populations. The model allows for some heterogeneity in both risk (perhaps genetic) and pathway (number of hits).”

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Multi-Hit Hypothesis for the Oncogenic Potential of mRNA COVID-19 Vaccines

Pathophysiologic Support for the Observation of "Turbo Cancer" after Vaccination
PETER MCCULLOUGH, MD
SEP 29

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By Peter A. McCullough, MD, MPH

As an internist and specialist, it is hard for me to believe a novel biologic product to cause heart, neurologic, thrombotic, and immunologic disease could also play a role in the initiation and acceleration of oncogenesis. In Western countries before the pandemic, the leading causes of death were heart disease 40%, cancer 40%, and death from other known causes (homicide, suicide, accidents, etc). The mRNA COVID-19 vaccines if proven to promote cancer, would then be implicated in rises in all-cause mortality being observed world wide.

In 1984, Sutherland and Bailer proposed the “Multi-Hit Hypothesis of Carcinogenesis:”

“A new multihit model of carcinogenesis is developed for use in evaluating age-specific cancer incidence rates in human populations. The model allows for some heterogeneity in both risk (perhaps genetic) and pathway (number of hits).”

They essentially said it takes multiple different hits or insults to cells and their genetic machinery to cause a normal cell to become cancerous. Forty years later, Sutherland and Bailer could not have dreamed about the application of their hypothesis to global mass genetic vaccination given every six months to a broad population, some with high risks for, or even with incipient cancer.

Angues and Bustos just released a paper on the Authorea preprint server that assemblies the evidence to date that both mRNA and the Spike protein work within human cells to cause changes that result in oncogenesis. The figure shown is consistent with a multi-hit hypothesis of oncogenesis after injection with Pfizer or Moderna.

More at this link:

https://petermcculloughmd.substack.com/p/multi-hit-hypothesis-for-the-oncogenic?
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Many Blessings,
CrystalRiver