This genetic information is important because up to 60% of the population have one or more of these genetic polymorphisms/mutations. Whether it is natural or caused by radiation, cell phones, EMFs, etc. the result is the same. If these issues are discovered at a young age, many chronic illnesses can be avoided in older age. I discovered that I have several of these mutations, some of which make it very difficult to detox the neurotoxins that are created by Lyme die-off. I thought I had pernicious anemia, which I do. However, I discovered that I also have a mutation which makes it impossible to store B12. Now that I am giving myself shots on a daily basis,what an improvement!! If a child, or a person, has methylation defects-MTHFR, it is impossible to detox with normal approaches. Needless to say, this leads to many chronic illnesses, and possibly chronic pain, in the future. If you have a chronic illness, it is highly likely that a genetic mutation/s is/are lurking in the background. If you don't feel inclined to get yourself tested, at least realize what a difference it can make in the future health of a child. I wish I had known much earlier what I know now. The world is not kind to our genetic make-up, but at least there is something we can do about it.
Lymerick
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FVL: Factor V Leiden. When factor V is found heterozygous, the odds of FVL’s having a clotting event in their lifetime is much greater than the someone without this mutation. Factor V Leiden is sometimes called Activated Protein C Resistance because this mutation leads to protein C resistance.
When pregnant, the uterus is fed with capillaries. So miscarriage is related to this particular mutation.
Remember, this is a gene where you will rarely see someone homozygous because it is usually not caught before a major clotting event occurs.
FVL can be serious even when heterozygous. So when someone knows that he or she has FVL, it is best for them to have their physician run platelets, C reactive protein and fibrinogen.

FVL is easily manageable.

These people are at higher risk of developing DVT especially when on hormone therapy or birth oral birth control.

Speak with your practitioner if you have any concerns.

F2 (Prothrombin 20210A) : Like FVL, it is extremely rare to find someone homozygous. Heterozygous people are at higher risk of developing a clotting event some time in their life when not monitored.

The F2 gene provides instructions for making a protein called prothrombin (also called coagulation factor II).

This gene is also related to miscarriage as the uterus is fed with capillaries.

Thrombin is also thought to be involved in cell growth and division (proliferation), tissue repair, and the formation of new blood vessels (angiogenesis).

2-5% of Caucasians and 0.3 percent of African Americans are heterozygous for G20210A.

It is important to be monitored throughout your lifetime if you have this mutation.

These people are at higher risk of developing DVT especially when on hormone therapy or birth oral birth control.

FUT2: The 3 major FUT2 genes that seem to cause problems are FUT2 A12190G, FUT2 G12447A and FUT2 G12758A. When these three SNPs are homozygous, these people cannot make H antigen.
Why is H antigen important?

SNIP